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Finasteride Cycling

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Finasteride Cycling

I’m seriously looking at Finasteride cycling to restore the sensation in my penis and hopefully gain some size. I want to get it right the first time without too much down time. Can those of you who have tried it please answer my questions, even if it didn’t work?

Propecia (1mg) or Proscar (5mg)?
What else did you take while on Finasteride?
Time on Finasteride?
Did you shrink while on Finasteride?
Successful (growth/permanent sensitivity increase if you had low libido) or unsuccessful?
Length of rebound phase
Any PE exercise during the rebound phase? Which exercises did you do?
Did you use additional DHT? How much and when?

Thanks!


BiffyStiffy

I can see the theory in the gains but not in the sensation. If I’m following you I think you want to take proscar for a while than stop hoping to get a DHT hormonal spike that you think may promote growth is that correct?


I haven't failed, I've found 10,000 ways that don't work. Thomas Edison (1847-1931)

DHT doesn’t work for growth after puberty. You’d be wasting your money. And I don’t see the sensation thing either.

I’ve been on finasteride for about five years (for hair loss) and my penis has not gotten smaller. It’s gotten larger with manul PE methods.

Westla is correct. There’s no point in trying to cycle finasteride to hopefulyl get a rebound in DHT. Blocking 5AR will result in increased estrogen production and this can lead to gyno and a slew of other problems. As mentioned, DHT alone fails to indice growth after puberty nad in some studies even during puberty. Studies showing DHT inducing penile tissue growth occurred in pre pubertal boys. Something during puberty causes the androgen receptors to become reduced and hence no penile growth even with excess DHT. Interestingly though the prostate will continue to grow in the presence of DHT. It for some reason does not experience this down regulation of AR. Fidn out what causes this downregulation of AR and you’ll be rich!

Yes Dino9x7, you’re right. But I’m not sure about taking Proscar because I’ve read that it takes several weeks for 5AR to upregulate again.

I know DHT alone would do little, this is why I want to see if Finasteride will starve my penis of DHT and cause a rebound phase when I go off it. Kevin Pezzi MD tried it and for him it worked. Both growth and increased sensitivity occurred.


BiffyStiffy

I used it, hated it. Listen to Westla.

>I know DHT alone would do little, this is why I want to see if Finasteride will starve my penis of DHT and cause a rebound phase when I go off it.

From this I gather you’re thinking that lowering DHT will cause androgen receptors to upregulate. It won’t.

http://www.mesomorphosis.com/articl…-regulation.htm

Pezzi is good at writing books on a variety of subjects. How well he actually knows any of them isn’t really clear. He may have been doing something else, such as a manual PE method, while taking the drug that actually gave him the results he attributes to finasteride.

UlcasterDropout, what did you use 1mg or 5 mg?

Hobby, the hypothesis is that DHT receptors will become sensitized rather than upregulated.

Westla, my primary aim is to resensitize my penis. Pezzi reported extreme sexual sensitivity during his rebound phase.


BiffyStiffy

Just 1mg per day.

Originally Posted by emitecaps
Westla is correct. There’s no point in trying to cycle finasteride to hopefulyl get a rebound in DHT.

I’m not aware of Finasteride users reporting rebounds either when they stop taking it. Perhaps the difference for Pezzi was the Yohimbine that he took before he began his rebound?

Originally Posted by emitecaps
Blocking 5AR will result in increased estrogen production and this can lead to gyno and a slew of other problems. As mentioned, DHT alone fails to indice growth after puberty nad in some studies even during puberty.

The increased estrogen is to be expected. Boys who fail to grow with increased DHT maybe growing/maintaining at full speed, and more DHT wouldn’t do anything. A factory working at maximum capacity would not have a higher output simply because more raw material arrived to be processed.

Originally Posted by emitecaps
Studies showing DHT inducing penile tissue growth occurred in pre pubertal boys. Something during puberty causes the androgen receptors to become reduced and hence no penile growth even with excess DHT. Interestingly though the prostate will continue to grow in the presence of DHT. It for some reason does not experience this down regulation of AR. Fidn out what causes this downregulation of AR and you’ll be rich!

After adolescence the DHT receptors possibly become desensitized, which means that unless the DHT receptors are resensitised, more DHT would be wasted in most cases.


BiffyStiffy

UlcasterDropout, did you regain any sensation/libido loss, and how long did it take?


BiffyStiffy

Also, how long did you take it for?

Thanks!


BiffyStiffy

It was a while ago, so my memory on it has decaded abit.

Took it for about 6 months. Loss libido in 3-4 months. Gradual.

Took about 6 months to come back to normal after stopping.

I’m not sure it’s a 100% of the original level. There was no blood tests or anything.

Originally Posted by BiffyStiffy
I’m not aware of Finasteride users reporting rebounds either when they stop taking it. Perhaps the difference for Pezzi was the Yohimbine that he took before he began his rebound?

The increased estrogen is to be expected. Boys who fail to grow with increased DHT maybe growing/maintaining at full speed, and more DHT wouldn’t do anything. A factory working at maximum capacity would not have a higher output simply because more raw material arrived to be processed.

After adolescence the DHT receptors possibly become desensitized, which means that unless the DHT receptors are resensitised, more DHT would be wasted in most cases.

What’s this desensitized receptor theory? There are no such things as dht receptors, only androgen and estrogen receptors. Testosterone and dht bind to androgen receptors and illict their effects. AFAIK a compound will bind to the AR or it won’t. If it binds it illicts an effect. It can’t bind only 30% and thus create 30% of the effect. In the bodybuilding world, the receptor sesitization theory is debunked. If it binds it illicts and effect, period, there is no degree to tge effect. The only thing that can determine any effect is the amount that binds, ie the number of receptors. Now if you are using a proscar or finasteride to block dht then none will be available to bind to the ARs. DHT is responsible for libido so if you have none then your libido is shot. Once you reintroduce DHT your libido goes up. This might be why people think there is this receptor sensitization.

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