Creep and plastic deformation of the Tunica Albuginea
I’m still going to read more about stretching and reconfiguration of the fiber matrix within the tunica albuginea even though I am 99% sure it is a dead end. I feel it is because the rare cases where the tunica albuginea was permanently stretched and erectile function was not destroyed the reports still indicate a decreased erection quality through decreased elasticity. The elastic feature of the inner columns of the tunica albuginea is what actually causes a stiff erection. If these structures are compromised by plastic deformation, then the erection will not function as it originally did. This means that if your penis can still obtain erections after some form force stretching it beyond capacity: priapism, excessive pump or stretching then it will never be as hard as it was initially because those internal structures have been altered so they no longer function as intended. They lost their elasticity, meaning the compressive force they would exert when stretched is now diminished and would be diminished relative to the amount it was stretched beyond initial capacity.
It will be argued that slow gradual changes in the tissue through creep is the mechanism through which PE is possible. I rebuttal that in the case of ligaments this is possible, but the increase in ligament length has not been shown in the literature to improve erectile length, only flaccid length. This was determined through post examinations and studies tracking lengthening phalloplasty (ligament cut followed by weights). In the case of the tunica albuginea I have not seen enough evidence regarding the remodeling of the fiber matrix to indicate that its elastic properties would be maintained. I highly doubt they would from what I have read so far even gradual stretching of ligaments (creep) will permanently deform them and cause a loss in their elastic properties. As for studies that counter the creep hypothesis in tunica albuginea: the two pumping studies showed no difference in girth and only the second study showed a difference in length which is attributed to the reduction in Peyronie’s curvature. Pumps and regular use of vasodilators are the best argument for creep for the tunica albuginea, stretching is confounded by ligaments being stretched, yet no studies that have been conducted noticed significant changes in penis size using pumps or vasodilators.
Saphenous vein grating does support creep for increasing penis size, but not in the tunica albuginea. A study found regular use of a pump following a saphenous graft applied to the tunica albuginea lead to greater increase in erect diameter. It was hypothesized that the veinous tissue stretched under pressure of the increased blood from the pump. A second study indicated a far higher rate of erectile dysfunction in the graft procedure, as high as 13% for one of the techniques (I believe their are two techniques: 1 for length, 1 for girth), and other complications such as decreases erection quality. So, creep with maintained function is possible in veinous tissue, but we already knew this from leg extension procedures. The issue is whether the tunica albuginea can maintain function after experiencing creep, the evidence so far does not support that conjecture.
I will finish reading the links Marinera sent me and will respond to the studies and ideas listed within those links.
Keep sending me stuff and I will go through it in my spare time.