The decreased size of the penis with positions is not a function of decreased tunica expansion in full erection. When you are in the sitting position with your legs at 90 degrees, you shift your pelvis downward, particularly the ischium where the cavernosa anchor to it (crux of the penis or root of the penis). This pulls your unexposed portions of the penis deeper into your pelvis and dragging some of your normally exposed penis with it. Your erection is not smaller, just less exposed.
I’ve been doing PE (including tunica stretches) and extender stretches for almost nine months with great success and rock hard erections. I have never had engorgement of my “outer layers’. When I have a full erection, my cavernosa surrounded by tunica is very hard. The over lying skin and connective tissue are applied close to the tunica.
I do not clamp or pump but if you cause a leak of fluid from blood vessels either by negative pressure (with pumping) or elevated vein pressure (clamping), the “increase girth” or “tumescence” you are seeing is edema and not real enhancement because it is temporary. To maintain this level of edema (subcutaneous edema), you will have to routinely pump or clamp. This edema fluid will either be reabsorbed by blood vessels or quickly drain via lymphatics. See my posts #4 and #6 on this thread for a physiological explanation of edemas formation and drainage:
I am not saying that clamping or pumping does not induce enhancement because it does. But unless I was (or still am) out to lunch and didn’t get the message, the overarching goal is to expand the capacity of the cavernosa by stretching the rigid tunica fibers to cause microinjury through plastic deformation and subsequent repair at a larger size. The subcutaneous edema is a side effect of the process and is temporary. Am I wrong here?
You need to explain to me how tissues are becoming more inelastic at smaller penis sizes or lengths.
It is the tunica that gives the restriction to further size at full erection. It is a connective tissue with relatively smaller amount of elastic fibers relative to the more rigid fibers. Its elastic component is what gives its stretchability and recoil initially (like the balloon). To get plastic deformation you must over come this elasticity because it tends to allow recoil to its normal shorter state. Therefore as you deform the tunica with stretches beyond the ability of the elastic fiber recoil, the rigid fibers take the forces. Therefore the tunica becomes inelastic at a longer fiber stretch length, not shorter. A balloon is completely elastic and has no rigid component, so your example is not completely applicable here.
Compliance of the tunica itself decreases as its fibers stretch and become inelastic. However after plastic deformation, fiber length (both longitudinally and circumstantially) will be longer. Therefore overall capacity of the chambers within the tunica will be greater so that at fully erect pressures (200 - 300 mmHg) the chambers will be longer and fuller, not less.
Your explanation as to a harder erection at a shorter length still doesn’t make sense to me. Can you or someone else elaborate to help me understand.