Microtears Result in Plastic Deformation
Tonight while searching for the correct spelling of the “Blachoe Ring”, while replying to another thread, I came across the Penis Enlargement Wiki. I decided to look at the information on the Wiki. I noticed the “Growth Theories” link, one which I have looked at in the past. I then clicked on “microtears”, because that is what I am finding occurs in connective tissue in order for permanent elongation to happen, according to the science behind physical therapy. In many cases, the term Microtruama is used. As I read the description of “Microtears” is see that the author states that plastic deformation and microtears are not one in the same, and that the two are clearly different.
Well, that lead me to this point. I’ve been wanting to debate whether or not plastic deformation occurs here on the forums, even though it has been debated over and over again. You see, I believe that both parties are right, to an extent, and I will clarify what I mean by that.
What misleads people is that they refer “only” to engineering mechanics when considering the definition of plastic deformation. In that case, the molecules of a material are stretched, creating permanent added length, but no new or extra molecules are added/created.
I also held this same definition of plastic deformation when I first discovered the term and the theory as it applies to PE. I reasoned that if my penis had 1 billion cells, and it permanently lengthened, then there is no possible way that it would still have only 1 billion cells. So, by that analogy, I disagreed with plastic deformation PE theory. But, I was not 100% sure that it did not occur in the ligaments. So, I kept the term in mind, even though I suspected the body would add new cells (extra replacement cells/more than the original amount) in the long run over a years time or so.
Now, lets take a closer look at connective tissue. According to information that I have found, about 47% of the penis is made up of connective tissue. Connective tissue is made up of collagen, and is very much like a ligament/tendon. I’m not sure how many members agree with me, but I believe that the connective tissue in the penis is the LIMITING FACTOR to developing gains.
Permanent elongation of a ligament “as a whole” is known as Plastic Deformation in biomechanics. Biomechanics is the science that orthopedic doctors and physical therapist refer to. So, to say that a ligament, or other form of connective tissue has become plastically deformed, is to say that it has become permanently elongated. Plastic deformation is not meant to explain what occurs on the molecular level in biomechanics. In both engineering mechanics and biomechanics plastic deformation is a result of MICROTEARS. The difference in engineering mechanics, the mircotears are not healed, and new molecules are not placed in the stretched areas, while in biomechanics, the body heals itself. The end result is new tissue, or extra collagen, which occurs by the process of inflammation, proliferation, and remodelling.
The elastic region represents the response of the parallel-oriented collagen fibrils to elongation, once the crimp has been removed. Elongation in thisregion is within its physiological capability and recovery is completely reversible. Elongation in plastic range will result in a degree of permanent tissue lengthening or if extreme, complete failure. Clinically this is the region where tissue damage occurs.
Notice, tissue damage occurs in the region permanent lengthening is achieved.
Now with the next bit of info. Since tissue damage has been created, the body must now heal itself. When the microtear or microtruama occurs, the contents of the cell spill out into the extracellular matrix, known as the ECM. This signals to the body that healing is needed, and this is the onset of inflammation. If the injury is large enough, swelling and redness are symptoms (this is in injuries such as sprains, and such a large injury is not a goal of PE).
Healing of the most connective tissue except bone, epithelium and endothelium occur by tissue repair. Thisis often accompanied by fibrous scar, which is the typical patching material for wound repair. Repair responses varyconsiderably in different injured tissues. After an injury, the healed tissue is never same as it was before. Tissue repair starts at 48 hours after injury and lasts up to 6 to 8 weeks, fibroblasts begin wound repair and collagensynthesis. Collagen has the property of gradually shortening when it is truly formed. This contracture occurs fromthe third week to the sixth month. New scar tissue will always shorten unless it is repeatedly stretched.Remodelling, also known as maturation, involves increased collagen density and organization, resulting inincreased tensile strength. This process begins as early as 3 weeks following injury and continues for a year or longer.
In that quote we see that healing of connective tissue is done by tissue repair. Remodelling, the “R” in IPR is mentioned in this quote, but inflammation and so on are also mentioned on that page as the process is outlined.
The rest of the penis is made of smooth muscle within the corpus cavernosa, and corpus spongiosum. This smooth muscle accounts for about 48% to 50% of the penis. The remaining 3% - 5% must account for the skin, veins, arteries, nerves, and capillaries. The structures of the penis mentioned in this paragraph or the ones that can be overworked. This is why high intensity routines require rest days. These structures are “more” responsible for engorgement of the penis during erection than the connective tissue is. This is the basis for negative PI’s occurring when these tissues are overworked. I am not sure of the process by which these tissues become permanently elongated. The skin, and the walls of veins and arteries are also made of of collagen, but I believe that mitosis may happen in the skin.
In closing, we now have evidence that plastic deformation is merely interchangeable with the words permanent elongation, and it is not used to describe what is occurring on the molecular level. Microtears/microtruama occurs on the cellular level in connective tissue, which is healed by the process of IPR, and results in permanent elongation, which is plastic deformation of the connective tissue.
The link to the site is: http://www.physiobd.info/articles/1…nflammation.pdf
Last edited by Kojack10 : 10-15-2007 at .