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Megalophallus through PE

Maybe it has to deal with the timeframe the cock is under extreme pressure. When you hear priapism its 4, 5 or even 6 hr timeframes of painfully enlarged erection.

Maybe this is why titleist marathon pumping of four hrs gets him gains.

Maybe it has to deal with the timeframe the cock is under extreme pressure. When you hear priapism its 4, 5 or even 6 hr timeframes of painfully enlarged erection.

Maybe this is why titleist marathon pumping of four hrs gets him gains.

Its for sure related to the timeframe.

And its inside pressure either by mechanical or chemical or both influenced.
Preventing outflow while giving more inflow. Theoreticly more similar to clamping.

Polymer is interesting cause he is drawing a connection to his kegel regime.

I know that I can with engaging my penis muscles hold more blood in it then without it.
Its ballooning. I feel there is more possibilities to do and increase it.

I basicly engage the IC muscle constantly while staying erect. Every now and then I kgel with the bc muscle which pushes more blood in which I hold immediately with an IC kegel hold. More then in a normal errection.
This then resembles “balooning”.
Now I can imagine these muscles going crazy and creating this balooning effect autonomous.
This then would probably be a mechanical priapism.
But I can hold such a ballooning expansion max only like 10 minutes for now without break.

I find interesting that he says the gain is immediate : after a few hours of priapism the penis is bigger. Why no scar tissue? I guess the answer is that cellular proliferation begins while the breaking point is still far, so it is never reached. The pressure goes high but pretty slowly. In a another similar case, a number of exams were performed and they found a hyperplastic tunica:
marinera - reduction surgery: penis skinning.

which suggests that there is a optimal level of intensity that, if prolonged for several hours, will give a longer tunica, without needs for recover. Which also implies that the mechanism at work is at least partially different then the one at work with most of PE exercises, where a higher intensity is applied but for shorter period of times - in this case, often gains are seen only after several days of rest.

..and with no fake expansion like with some PE.
Pumpers who can and do it for hours get no priapism especially not the extreme pumpers.
The pumping gainers mostly advice to long low pressure pumping.

The more in less out priapism is gradually increasing the expansion?
Polymer is it gradually getting bigger or staying at a certain size. (and how is it correlated like with pain?)

So I assume the pressure is all internal. Smooth muscles filling with more and more blood. Pressuring each other and the limits of the tunica without any chance to rebound elasticly.
Would even some short pauses during a priapism stop the enlargment cause elasticity is temporary back?

What is hurting? Everything? The tunica? When I edge for a long time and then cum and restart PE immediately I feel pain all over. PE after ejaculation
There is something chemicly happening. Like “old NO” or..
This process could happen over and over during a priapism keeping a chemical process active.

This paper:
http://webcache.googleusercontent.c…e&ct=clnk&gl=de
named Chapter 2 Normal Erectile Physiology

under the bullet point:
Hemodynamics of Erection

explains rather good how in and outflow is going on. I guess somewhere there the muscles throough whatever impulse “cramp”/engage to shunt the dick off.

How could the tunica weaken after continious(increasing or stable) pressure from inside?
Quote:

“We propose an alternative mechanism for this sequela. The increase in penile circumference follows a sudden and permanent loss of elasticity of the tunica albuginea, brought about by a particularly intense priapism and a very engorged organ. The loss of elasticity of the tunica albuginea releases constraints on the corpora cavernosa, which then expand like a sponge. This expansion, helped by some subtunical venous impairment that is secondary to the remaining elasticity of the stretched tunica, results in pooled, deoxygenated blood in the corpora cavernosa consistent with our BOLD-MRI findings. BOLD-MRI defines the presence of deoxygenated blood, eliminating the need for an invasive procedure.”
http://www.ncbi.nlm.nih.gov/pubmed/…4?dopt=Abstract

No deoxinated blood in pumping I suppose.
The sudden loss shows it can happen probably sometime when the blood is deoxygenated to a certain level.
As with a “non ischemic” it probably still isnt totally deoxygenated as fresh oxygen follows which then decreases the chances of a defunct penis.
Of course experementing with this is highly dangerous and can result in a defunct Penis.
But in its mildest form resembles many intermediatery PE exercises performed by conditioned exerciseerss.
There are some examples of people pushing their limit regarding oxygenation that had some good gains and documentation. Like Redzulu. Or big girtha. They for sure created internal pressures for a long time. They all had a long conditioning before venturing into such extremes.

I would suggest for anyone getting out of control priapisms to immediately visit a doc to relax the situation. The chances of a defunct penis are probably too high to not get to the doc..
There might be the possibility to get control over it with training the pelvic floor if the high in flow isnt created/originated by lets say the brain or spinal cord…

hope someone chimes in and this has some truth in it ..

Is the inflow continiously increasing the pressure and size(and maybe correlated pain?) of the organ? Or is it at some point stable? Polymer ,can you recount that?

They are supposing that it happened through a sudden loss of elasticiy, nothing is backing that up. They didn’t perform any specific exams either there, differently from the paper in the penis skining thread, linkd above. Polymer’ case doesn’t suggest a sudden lose of elasticiy if I am understanding it. I think a sudden loss elasticity would make more likely a penis rupture, never seen in megalophallus’ cases.

Having deoxygenated blood cause necrosis of tissues, resulting in ED, so I don’t think it is what is happening in the cases reported here.

I don’t know if I can remember where I posted it, but there was a very old report of two megalophallus cases, with b&w pics, as a consequence of wearing a harness similar to a leg-strap ADS, in the first case, another one as a consequence of a trauma.

Originally Posted by marinera
Having deoxygenated blood cause necrosis of tissues, resulting in ED, so I don’t think it is what is happening in the cases reported here.


It might be a side symptom but not the trigger of the tunica penis increase.

Quote
I don’t know if I can remember where I posted it, but there was a very old report of two megalophallus cases, with b&w pics, as a consequence of wearing a harness similar to a leg-strap ADS, in the first case, another one as a consequence of a trauma.

Most trauma like extremes stretch or hit will even in their smallest forms create a pelvic floor response.
Its what I believe happens at least in part(symptom or trigger, again?)
I believe it causes the muscles, or at least aid, holding´outflow to the cc back while allowing inflow. through other channels. A priapism is essentially a balooning (and the crazy Dr. Lin is right after all with his stick making happy woman ).


Last edited by memento : 03-05-2015 at . Reason: Removed excess unfinished quote

BUT hypoxia is probably the main cause in the case of sickle cell anemia induced priapism..
But in it could jsut be the symptom of to different states.
1.holding the blood in the penis through whatever mechanism: more hypoxy blood even with oxygenated blood.
2.Sickle cell anemia: hypoxy as a condition of the condition. Thus high potential for hypoxy i nthe penis(under prolonged errection)

Could it be the Tunica needs oxygen to hold its structure?

Also: bad blood circulation and maybe no oxygen at all to a part of the tunica = scar tissue(peyronies disease)

Originally Posted by marinera
This also is related
marinera - Loading, lengthening, healing.


” The exact mechanism of priapism is not well understood.

However, in sickle cell disease, it is believed to be due to blockage of venous

drainage by sickled cells. “

______________
Boy had errections several days at 17. I suppose it was not as intense as what some others report like polymer.


Last edited by dickerschwanz : 03-05-2015 at .

Originally Posted by marinera
Found
Hypertrophy Of Penis

. Some three weeks before presenting himself at the hospital while attempting to turn professional somersaults with the organ strapped between his legs the foreskin was wrenched, bruised, and slightly chafed;- this condition was followed
by inflammation and oedema of the prepuce, with suppurating periadenitis of both groins.

_________
Prolonged BTC stretch with errections and many bends and other stuff.

Can somehow imagine this guy with his strap. brutal acrobatics with the girls etc. with his horsecock dangling Must be annoying. lol

This has shed a lot of light on my pumping routine. I don’t think it’s advisable for all, but certainly works after conditioning.

Thanks for all of your fact finding guys!


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