I have long suspected that elasticity-to-plasticity is probably the most significant determinant in a PE’ers progress - more so than “LOT” or starting size, or any other variable.
In wantsmore’s thread, “The Upper Limit of PE: Does it exist?”, I responded in
“Yes, this is precisely what I mean. Gains come as elasticity is exhausted (& plasticity occurs). Ergo, those with more elasticity (“growers”) have more gain potential - although a lot of elasticity can be tough to combat. While those with less elasticity (“show-ers”) might respond quicker (i.e., achieve plasticity sooner), they would theoretically have less potential.”
Even though some expressed skepticism regarding this idea, I had recently began a number of web searches (with the thought of finding/developing a topical muscle relaxer, with the hope of combating the limiting aspects of the tunica regarding PE) when I came upon the following…
“Priapism is a common complication of sickle cell anemia. We report a little known sequela of priapism: painless megalophallus, with significant penile enlargement. The patient had had an intense episode of priapism 9 years previously and his penis remained enlarged. Blood oxygen level-dependent magnetic resonance imaging revealed enlarged, hypoxic corpora cavernosa. Megalophallus probably resulted from permanent loss of elasticity of the tunica albuginea due to severe engorgement during the episode of priapism. This sequela needs to be recognized by physicians because no intervention is necessary and sexual function seems to remain intact.”
While its been known for quite some time that priapism can cause megalophallus, I was surprised with this description, for several reasons:
(1) A little known “sequela” of priapism may be painless megalophallus
(2) This type requires no medical invention
(3) Sexual function seems to remain intact
(4) In this case, only one episode of (severe) priapism resulted in this megalophallus
(5) His enlargement, having lasted 9 years (& counting) is most likely permanent
(6) They cite that the “Megalophallus probably resulted from permanent loss of elasticity of the tunica albuginea…”
While this proceeded dramatically (unlike typical PE), I would imagine that as his erection persisted (severely), the pressures against the tunica began to weaken (short-term) the structure, which allowed the erection to swell above previous size. Obviously, this could only happen so much before a severe injury occurs (which did not occur to this man - his sexual function remained intact). I would imagine that his girth probably swelled to the point right before a blow-out, then his erection(s) subsided.
But because the severe engorgement resulted in the permanent loss of elasticity, he never really went back to his original flaccid size…in a sense, he became the “ultimate” show-er (but more than that, because the very name “megalophallus” implies that there was also actual enlargement, and not merely greater flaccid size).
Obviously, none of us would want sickle cell anemia. Nor should any of us trifle with priapism, or make attempts to artificially induce this dangerous condition, I believe that the article at least implies 2 things:
(1) Greater size DOES result when penis tissues lose their elasticity.
(2) These types of gains (“cemented” gains occuring due to plasticity) appear permanent - or very nearly so [suffering only, perhaps, very minimal losses].
I don’t think that expanding the spongy tissues is the tough part at all, it’s overcoming elasticity. I think a good supplement such as ginseng (or some other vaso-dilator) will promote all the bloodflow you need. The next step is in overcoming the tunica.