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For Science minded PE'ers

Yes, there is a limit to which you will no-longer receive any gains of significance. The capacity for elongation is greater for any incremental size of the tendon. Much like comparing the capacity of a 2 inch rubber-band verses a 6 inch rubber-band, the six incher has inherently greater stretch capacity.

Originally Posted by Tomba
The loss of some girth with increasing length is a natural result and it occurs because stretching tendons also results in stretching arteries and veins. As this physiology elongates you will have a slight reduction in the diameter of your blood inlets thereby affecting the flow characteristics of engorgement.

However many times I read the above, I cannot make that model work in my head. Blood inlet diameter would be an engorgement rate limiting factor, not a volume capacity limiting factor. If it takes you 1 minute to get a full erection rather than 2 minutes then all is well and good, but a 100% erection is just that. Drip by drip you can fill the ocean.

Originally Posted by Tomba
Much like comparing the capacity of a 2 inch rubber-band verses a 6 inch rubber-band, the six incher has inherently greater stretch capacity.

The difference between that analogy and our situation is that our elastic band is part of an intelligent organism that has the ability under the right conditions to cease being a 2” elastic band and evolve into a 6” rubber band. This is demonstrated in every living thing around us every day. Judging the capacity of a 2” rubber band is not the same as judging its potential. If that were the case then Einstein would have remained a patents clerk all his life.


Last edited by Shiver : 09-16-2004 at .

There is a pressure gradient that you are not factoring in your model. As you have a reduction in diameter you have to have a equal adjustment in pressure in order to blow up the balloon to the same dimensions.

Medical journals have documented this trait in tendon elongation; although it is organic matter there is an elasticity coefficient built-in. The analogy holds true.

Originally Posted by Tomba
There is a pressure gradient that you are not factoring in your model. As you have a reduction in diameter you have to have a equal adjustment in pressure in order to blow up the balloon to the same dimensions.


No.
In any system, the pressure is constant throughout a liquid, regardless of the diameter of any pipe carrying the liquid.
As Shiver has said, you will have a decrease in the velocity of the liquid due to the size of the pipes (the rate at which an erection may be achieved).

Also, I still can’t figure out who the size of an artery feeding the penis effect the size of the erection. The shape and size of an erection is ultimately dictated by the shape and strength of the tunica (all other things such as erection quality, ligament attachment points, etc, being equal).

To say anything to the contrary is plain incorrect. Tomba, you may have gained what you claimed by using the exercises you champion, but I believe it is certainly not due to your stated theories.

Shiver, your trellis analogy was excellent.

Originally Posted by Andrew69
Whenever I have made a significant length gain, it has been at the expense of some girth. I have been lucky enough to keep my length gains coming on a fairly regular basis, but when I do target girth, my length gains slow or even stop.

Lam, a guy who used to post at PE Forums, reported losing girth when gaining length. Then his girth would catch back up. It happened in cycles.

Personally, I haven’t lost any girth that is locked in when switching to a length-only focus. One girth gain is still somewhat puzzling though. I did only squeezes and other girth stuff for a couple months. No stretching or hanging. I gained a little in the first few weeks, then no more.

Then I switched over to hanging but continued some very mild girth work - a few squeezes here and there and some wrapping. I not only gained length that month but also picked up some girth over the whole shaft. Why did I gain girth under the circumstances? I don’t know. Could it have to do with the hanging exerting force primarily on bonds in the other direction, breaking or rearranging the crosslinks that had been established from all my earlier squeezing?

Originally Posted by Andrew69
No.
In any system, the pressure is constant throughout a liquid, regardless of the diameter of any pipe carrying the liquid.
As Shiver has said, you will have a decrease in the velocity of the liquid due to the size of the pipes (the rate at which an erection may be achieved).

Also, I still can’t figure out who the size of an artery feeding the penis effect the size of the erection. The shape and size of an erection is ultimately dictated by the shape and strength of the tunica (all other things such as erection quality, ligament attachment points, etc, being equal).

To say anything to the contrary is plain incorrect. Tomba, you may have gained what you claimed by using the exercises you champion, but I believe it is certainly not due to your stated theories.

I agree. The usefuleness of the physics relies on the acceptance that a baloon/rubber band is a valid working model. I would say something like a tyre (tire? :) ) would be a better working model. The wire or threads in the tyre are the target, not the rubber that we see. Anyone who’s ever had the experience of a blowout would readily accept that it is not the diameter of the inlet valve that caused their experience.

Quote
Then I switched over to hanging but continued some very mild girth work - a few squeezes here and there and some wrapping. I not only gained length that month but also picked up some girth over the whole shaft. Why did I gain girth under the circumstances? I don’t know. Could it have to do with the hanging exerting force primarily on bonds in the other direction, breaking or rearranging the crosslinks that had been established from all my earlier squeezing?

I would say that is very likely the reason.

Shiver,

In your original post, what exactly do you mean by “crosslinking?”

Thanks!

Originally Posted by ModestoMan
Shiver,

In your original post, what exactly do you mean by “crosslinking?”

Thanks!

The dictionary definition:
http://dictionary.reference.com/search?q=crosslinking

There are however many variations and causes of crosslinking (many of which I do not yet fully understand) which makes the whole thing very confusing. DNA to protein linking for example is something to be avoided, which in an ideal world would trigger apoptosis to avoid mutations. That’s not to suggest that PE causes that in any way more than other of lifes activities.

Some of the variations are covered in this article:
http://www.vrp.com/library/732867.h…ary/726737.html

http://www.redwingbooks.com/html/ca…/TL_Extract.pdf

“When the inflammatory process is prolonged, it causes problems for the ligament or tendon that is healing. Continued inflammation leads to excessive adhesion, fibrosis, and scarring. There will be inapropriate cross-linking of the fibers which contributes to dense, inflexible tissue. The developing fiber orientation is often random and scattered (not parallel).”

Well, just how do you know if your penis is eaten up with fibrosis? How could one gain? Mine mustn’t be now cause of some gains I’ve been making, but I would like to know if I start wasting my time stretching unstretchable tissue!

Originally Posted by hobby

Lam, a guy who used to post at PE Forums, reported losing girth when gaining length. Then his girth would catch back up. It happened in cycles.

I can report the same, although I don’t have measurements to back up my claim, I do have pictures. It will be a while before I worry about girth, I’m not sweating it.

Alice

Originally Posted by Shiver
http://www.redwingbooks.com/html/ca…/TL_Extract.pdf

“When the inflammatory process is prolonged, it causes problems for the ligament or tendon that is healing. Continued inflammation leads to excessive adhesion, fibrosis, and scarring. There will be inapropriate cross-linking of the fibers which contributes to dense, inflexible tissue. The developing fiber orientation is often random and scattered (not parallel).”

I’m not a technical kind of fellow obviously, but would heat cause any changes?

Starter:
I don’t know the answer to that one. I think it’s a given that we all get fibrosis to a greater or lesser degree, which is natural but will put a practical ceiling on our gains. I’m toying with the idea of making my own capsules that might help reduce the build up. It will certainly include carnosine and carnitine, and hopefully ALT-711 (supplier permitting). Much more to learn though before devising a formulation, and it’ll be hard to know if it does anything at all for a while since PE is such a marathon.

Alice:
Heat in the right temperature zone should allow you to quickly manifest the maxumum extensibility from the existing tissue (in my case about 1 week). Anything after that is old school hard work damage & repair cycle.

Well, the rubberband theory sounds pretty sound for the most part, there is another factor!

A rubber band is not able to repair itself and increase in size, it is limited! If we can increase the size of the tissues and they can repair themeselves without scar tissue, then they can be stretched again. I believe from all that I’ve read in the past, that scar tissue is not a direct result of micro trauma, but exclusivly etreme trauma. I believe the same kind of elastic tissues that are micro torn will bridge the gap! There is probably some competion between elastin and collegan, if the gap is to wide then collegan gets the pleasure, if not, good ol’ elastin to the rescue! But what do I know.

Tomba, could you provide us a link to other threads concerning increasing the pipes…bloodflow capacity? Thanks. Good thread, guys!

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