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Focus Your PE and Gain! TGC Theory

Non sequitur: Sometimes I wonder whether the majority of length gains are actually not in the tunica or the smooth muscle but rather in the ischocavernosus and bulbocavernosis muscles—skeletal muscles. I think Penismith also posted about this a while ago. Curiously, I can tug back much further now than I could when I first started PE, and the more I gain, the more it seems I can tug back.


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Originally Posted by ModestoMan
Non sequitur: Sometimes I wonder whether the majority of length gains are actually not in the tunica or the smooth muscle but rather in the ischocavernosus and bulbocavernosis muscles—skeletal muscles. I think Penismith also posted about this a while ago. Curiously, I can tug back much further now than I could when I first started PE, and the more I gain, the more it seems I can tug back.

But that may simply be a consequence of doing your exercises (kegels).


Later - ttt

Originally Posted by Iguana
Thanks tick, I was hoping you would join in. Very interesting find! Did you find anything related to similarities/differences in composition?
Is there any reason to believe that penile smooth muscle cells would not react similar to vascular smooth muscle cells autocrine or paracrine growth
Mechanisms?

Is this information available on-line?

Thanks!

The paper I found can be downloaded, M.E. DiSanto: Corpus cavernosum smooth muscle physiology.. J. Andrology 2003;24:S6.

I am not sure about the mechanisms; there is no other organ with this distribution of smooth muscle as far as I know.

As for mechanisms, it would at least be appropriate to compare with other organs where smooth muscle plays a major rol; urinary bladder comes to mind - in patients with outflow obstruction - prostate enlargement - the smooth muscles thickens a lot-I guess as a response to increased pressure required to void against the resistance of the prostate.


Later - ttt

Originally Posted by stagestop
Hi TTT,

With the architecture of the smooth muscle being as you described, do you have a thought about the ability of the various growth factors to increase this muscle size?

No idea at this point. However, all of us can search for smooth muscle - just anywhere - and growth / growth factors. And we have a lot more organs to compare with.


Later - ttt

Originally Posted by ticktickticker

But that may simply be a consequence of doing your exercises (kegels).

True, but I haven’t done that many :) .

I mention this possibility half tongue in cheek. I’ve always wondered whether the tunica extends fully over the ischiocavernosus muscles or whether it thins or vanishes altogether as it passes over (or through) those muscles. If it thins or vanishes, the area around IC muscles could simply be the weakest link in the lengthwise chain of the penis and possibly the easiest to elongate.


Enter your measurements in the PE Database.

Probably posted before, but a good illustration of the penile skeletal muscles.

http://www.webmd.com/NR/rdonlyres/C…8AB29BD9CE.jpeg

another one of the tunica:

http://www.andrologen.info/andros/z…es/web_Fig2.jpg

Another one…

http://images.google.com/imgres?img…%3Doff%26sa%3DN

Scroll down for some really good diagrams under “root of the penis.”


Let me tell you the secret that has led me to my goal: my strength lies solely in my tenacity.

Louis Pasteur


Last edited by Iguana : 03-25-2008 at .

Originally Posted by ticktickticker
As a consequence, they probably cannot be equated with the smooth muscle in blood vessels, and truly I know of no other organ in which the smooth muscle is arranged in such a manner.

Great observation, t3. Good to have you input here.

From my research, it appears the smooth muscle in the penis is like no other type of smooth muscle (Disclaimer: I could be off here, as for every journal I’ve read, there’s at least 25 I haven’t). For one, the smooth muscle spends most of it’s time in a relaxed state and only contracts when you have an erection.

As a side note, I think it’s fairly clear that smooth muscle grows to the stress of PE. If we think about the other tissue in the penis that has smooth muscle in it (blood vessels), we notice that these often grow and become more visible due to PE. Although the VSM (vascular smooth muscle) is different from the CSM (corporal smooth muscle; i.e. the smooth muscle we’re talking about in this article), the VSM still appears to enlarge, and hence why we often get enlarged and more prominent veins due to PE.


TGC Theory | Who Says The Penis Isn't a Muscle?

"To leave the world a better place, to know even one life has breathed easier because you have lived is to succeed." - Emerson

Originally Posted by remek

For one, the smooth muscle spends most of it’s time in a relaxed state and only contracts when you have an erection.

Didn’t you mean this the other way around? Penile smooth muscle is generally contracted and relaxes when you have an erection.

Originally Posted by remek

As a side note, I think it’s fairly clear that smooth muscle grows to the stress of PE. If we think about the other tissue in the penis that has smooth muscle in it (blood vessels), we notice that these often grow and become more visible due to PE. Although the VSM (vascular smooth muscle) is different from the CSM (corporal smooth muscle; i.e. the smooth muscle we’re talking about in this article), the VSM still appears to enlarge, and hence why we often get enlarged and more prominent veins due to PE.

I don’t know that “it’s fairly clear that smooth muscle grows to the stress of PE.” A better statement might be that it is “plausible” and not inconsistent with the response of similar tissues in the area.


Enter your measurements in the PE Database.

I think the corporal smooth muscle is closer related in behavior to blood vessels than we think. I think this is a reason it grows more CC grows from the stress of PE. Like when you run or jog or exercise you can actually grow bigger and more blood vessels. I could be wrong. I finally hit 8”NBP Saturday. I was applying this theory to PE. I started using the principles of Remeks/Iguana’s theory. I was always close to 8”NBP say around 7.8” and once in a great while 7.9”. So I will continue to see if I get more substantial gains from it.


Speak softly carry a big dick, I'm mean stick!

Originally Posted by kingpole
I finally hit 8”NBP Saturday. I was applying this theory to PE. I started using the principles of Remeks/Iguana’s theory.

Congratulations! Sorry to interrupt kingpole but what exactly you did to achieve it? I remember you from pumping forum-how did you apply Remeks/Iguana’s theory to your PE workout if I may ask?

Way to go you guys. I appreciate the hard work and the eloquent presentation of what appears to be a great idea.

Regarding measuring BPSFL:

I pull very hard, and at completely flaccid. I do 10 one second pulls basically as hard as I can stand, and use the last measurement.

I know there are threads explaining how to measure BPSFL, but these sometimes differ slightly, and I thought you may want to set a standard way to measure. Basically it amounts to how hard one should pull, and maybe for how long. I apologize if you have already done this somewhere, I am trying to cut down my Thunders time.

I also have recently been looking at a “just the slack out” measurement. This is about 5 quick, very, very light jerks just out to the end of the “slack” in my flaccid. Bone pressed. So this is not “stretched” hardly at all. I don’t know if this is how some of your participants are measuring, I haven’t taken the time to look at the thread.

Another flaccid measurement I have been looking at is the same as the “just the slack out”, but at say 20% erection. Plump, but still very flexible, bone pressed. I have found that this measurement is almost exactly the same as my BPEL, and that it seems fairly consistent.

I only post these ideas/measurement techniques as food for thought. Perhaps they will be useful as you develop your ideas further.


Sept. 4, '07: BPEL 6.875 inches, EG widest 5.25

Goal: Double digits

Originally Posted by chronostone
I am trying to cut down my Thunders time.

:)


"Debate the idea..."

Originally Posted by ModestoMan

True, but I haven’t done that many :) .

I mention this possibility half tongue in cheek. I’ve always wondered whether the tunica extends fully over the ischiocavernosus muscles or whether it thins or vanishes altogether as it passes over (or through) those muscles. If it thins or vanishes, the area around IC muscles could simply be the weakest link in the lengthwise chain of the penis and possibly the easiest to elongate.

Tunica is attached to pelvic bones.


Later - ttt

Originally Posted by remek
Great observation, t3. Good to have you input here.

From my research, it appears the smooth muscle in the penis is like no other type of smooth muscle (Disclaimer: I could be off here, as for every journal I’ve read, there’s at least 25 I haven’t). For one, the smooth muscle spends most of it’s time in a relaxed state and only contracts when you have an erection.

Sphincter muscles (anal and others) are similar: always contracted to avoid leakage, relax for defecation and anal sex.


Later - ttt

Originally Posted by stagestop

IGF-1 (insulin like growth factor 1) causes cell division in the cells that it touches (simple explanation). When you inject it into the penis it is then in the CC, and not the tunica. Growth is caused partially by cell division. So, as you continue with the IGF-1, you get a bigger CC. As the CC is inside the tunica, which takes much longer to respond, you are basically trying to get 10 gallons into a 5 gallon hat. The cell division is all in the CC and nothing touches the tunica, so the CC grows (mostly in circumference) When an erection occurs, you can feel the tension and it has a dull ache to it. This is why I mentioned the pain aspect in my previous posting. This is a clear sign that one has to stop with the IGF-1 until the tunica can catch up to the existing CC expansion.

I just ran across this quote from stagestop here: stagestop - Chemical PE progress report It seems very similar to the idea behind this theory, as he also stated here:stagestop - Focus Your PE and Gain! TGC Theory

The difference being that chemicals are the catalyst for a growth response vs stress placed upon this tissue. His observations/experience seems to me to add credence to what is being postulated here, being that, the smooth muscle can grow and expand at a rate different that the deformation of the tunica creating more pressure inside the penis. This appears to be consistent with the assumption that more smooth muscles equates to harder erections.


Let me tell you the secret that has led me to my goal: my strength lies solely in my tenacity.

Louis Pasteur

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