Thunder's Place

The big penis and mens' sexual health source, increasing penis size around the world.

Ligament ache pain, good or bad?

Why are you editing those posts?

Yes I gained girth on my whole shaft from hanging. Are you implying that I’m lying about that?


Hanging through the year 2012. Check my log.

Originally Posted by marinera
:rolleyes:

Are you still pulling out your inner penis?


No, I’m deforming the tunica :cool:


Hanging through the year 2012. Check my log.

Yep. Like Bennet8. He has pics.

Somebody time ago was complaining why the hanger forum looks like a dead place. Reading the posts of Holy Hangers gives an hint.

What about Bennet8? He made gains from stretching. So? There are also many more pics on the pics forum.

There are plenty of hangers on other PE forums. Reading “facts” from the PE Gods here, it’s not a surprise that many guys moved to other forums.


Hanging through the year 2012. Check my log.

Okay, I won’t post to newbie forum anymore. I’ll stay in the hangers forum. I don’t want any troubles or fights here.


Hanging through the year 2012. Check my log.

Originally Posted by conjunction
Look at that enthusiasm, screen actors guild approves

Hahaha, well, I just wanted to share my enthusiasm!

Originally Posted by mister007
I don’t know if it’s accepted idea. Seems like that here in thundersplace.org hanging is curse word for some.

Apart from the method used (in your case hanging) do most people agree that when your not gaining from BTC anymore, you should focus more on SO and or OTS?

Btw i totally agree on the logic with witch you approach the whole angle thing. At some point (after ligament gains are maxed in a current state of tunica+inner penis) when gains have started coming from tunica and or inner penis, one would have to go back to BTC to accomodate the growth.

To marinera and mister007: You both have valid points and you have both made great gains. I respect both of your views, I just wish you could debate your opinions in another thread, as they aren’t strictly related to my question(s) :) please :)

Im currently reading the links provided by firegoat about how collagen actually deforms to elongate ligaments and i’ll probably have more questions relating to my original question when im done with that.

(Ache has gone down btw, not completely gone. Have been doing 5 min manuals + 120 jelqs daily)

Yeah, that’s what I was repeating. ;)

Originally Posted by john.bigson
Apart from the method used (in your case hanging) do most people agree that when your not gaining from BTC anymore, you should focus more on SO and or OTS?

Btw i totally agree on the logic with witch you approach the whole angle thing. At some point (after ligament gains are maxed in a current state of tunica+inner penis) when gains have started coming from tunica and or inner penis, one would have to go back to BTC to accomodate the growth.


There seems to be different opinions. Some people stretch/hang/whatever in many different angles. Some people focus on one or two angles until the gains stop.

At least in the hangers forum it’s accepted idea to hang lower angles first then go to fulcrums and upper angles. This is mainly because hanging seems to take lot of time and who has time to hang all the angles?

Seems like some manual PE guys also start doing fulcrum after newbie gains are stopped. Some guys move to more advanced PE after newbie gains. Some gain well with basic stuff and never need to go for advanced.

Sorry for turning this thread into a pissing match earlier. I will not post anything here anymore until you quote and ask me something.


Hanging through the year 2012. Check my log.

Originally Posted by firegoat
Hope this helps a little. firegoat - Loading, lengthening, healing.

This too: /forum/showthread.php?p=1239100#post1239100

Ok, so i’ve finally read and understood (i think) one of the quoted reports, namely “Intrinsic fibroblast-mediated remodeling of damaged collagenous
matrices in vivo” and i’ve come to 2 conclusions.
1: Based on the following quote:

“For instance, examination of
collagen microstructure with scanning electron microscopy
after subfailure damage revealed ruptured collagen fibers
and fibrils, and fiber and fibril dretractionT appeared to have
taken place (Fig. 8). If proteinases are to clear debris and
remove damaged tissue after injury, it is unlikely that the
initial gap between ruptured fibers and/or fibrils would, at
that point, decrease. As such, if the tissue gap is present
when new tissue is dfilled inT, the repaired fiber or fibril
would be longer than its pre-injury length and as such more
lax. It is unclear whether long-term remodeling would
resolve this issue or not, although examination of remodeling
ligaments following subfailure injury revealed substantial
laxity remained 2 weeks post-injury”

small partial tears do occur. This is what i want.

2: Small partial tears fall in the category “grade II sprain”, or subfailure, and when such damage occurs there is “little or no inflammation”.

Summation and course of action: i want to keep my stretching just below the inflammation point. Why? Because when inflammation occurs, the (wanted) type 1 collagen fiber dominated repair is replaced with type 3 collagen fiber dominated repair. Type 3 is what is known as scar tissue. Scar tissue in the penis itself can cause ED problems. This is not what i want, even if scar tissue can be replaced through the years with type 1 fibers.
Scar tissue in the suspensory ligaments, as i see it, wont cause ED, since those ligaments do not inflate to cause erection.
Inflammation is painful, so i’d do myself a favor by going for the subfailure type PE, even in the case of the suspensory ligaments….UNLESS causing large enough ruptures for inflammation actually makes my gains faster or bigger. This “asterix” only concerns the susp. ligaments though, not the rest of the penis, such as tunica.

Further questions: Could tears involving major inflammation and scar tissue dominated repair generate faster and or bigger gains in the suspensory ligaments?

I think you already answered your own question.

Originally Posted by marinera
I think you already answered your own question.

I did? I must be missing something then. Please explain!

If inflammation cause scar tissue production, how that can lead to faster or bigger gains?

Originally Posted by marinera
If inflammation cause scar tissue production, how that can lead to faster or bigger gains?

I don’t know. But im not a physician and i know that sometimes what I think is logical, the facts of biology do not agree with.

I was pondering the idea that with large damage due to higher stress, the gaps between the ruptured fibrils would be longer. When the scar tissue is formed it connects over longer gaps, ergo quicker gains. Theres the question also of how many fibrils that rupture. For a full elongation, from a mechanical viewpoint, in the end all fibrils need to rupture and be elongated. With higher stress the rupture of fibrils are performed at a higher rate (but not all at once since i dont want to snap them in half, wich would probably require reattachment surgery). Those are the ideas.

Im sure there are things i do not know of, like for instance perhaps scar tissue has a tendancy to shrink? It takes longer to be formed? (Although i think that the report i read implied that it responds at the same rate as type 1 fiber formation) Other things that speaks to its disadvantage? Please enlighten me, i dont like this introverted luke skywalker approach to learning :D

And i repeat - i only consider this for the suspensory ligaments, due to the ED risk of having scar tissue in the body of the penis.

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