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Length exercises....

Length exercises....

Hey there, TP. I was wondering if you do ONLY length exercises, will you lose any girth?

Doesn’t seem that way. I haven’t read of anyone that did only stretching or only hanging complaining about loss of girth, as I recall.


regards, mgus

Taped onto the dashboard of a car at a junkyard, I once found the following: "Good judgement comes from experience. Experience comes from bad judgement." The car was crashed.

Primary goal: To have an EQ above average (i.e. streetsmart, compassionate about life and happy) Secondary goal: to make an anagram of my signature denoting how I feel about my gains

Well i saw one person complain, so that got me wondering .

ADS devices claim that they increase the girth. If that’s true is unlikely to lose girth from stretching.


Starting 06/2010 with BPEL 5.25'', EG 5''

Goal: BPEL 6", EG 5

Long term goal: 7x6

Hello km21, I asked exactly the same question yesterday, here you have a link to some other responses (my thread).

Can I lose girth with stretching only routine

Hope this is helpful. Good luck.

Europeanguy

Originally Posted by mgus
Doesn’t seem that way. I haven’t read of anyone that did only stretching or only hanging complaining about loss of girth, as I recall.

Although your question was already answered by mgus, I’ll elaborate a bit:

Lots of guys do penis weight hanging to increase base girth (and counteract the baseball bat effect). Extender / Stretcher use and manual stretching can increase base girth as well (but both seem to give less base girth than hanging is capable of adding). Keep in mind, not everyone gains base girth with each of these exercises; some do, some don’t.

I’m not aware of any length specific exercise that can give mid-shaft girth . However, it does seem every length exercise can give a little bit of girth at least at the base, while not all girth exercises seem capable of giving length.

In other words, not only will you not lose girth, you may (or may not) gain some extra base girth with length exercises.

As far as I know, the sole length exercise that gives base girth is hanging. Extenders do near nothing for girth, except a minimal gain right behind the glans reported by those who use noose-style extenders.

Here’s one example:
/forum/en/showthread.php?t=130031

Originally Posted by bohm
Gained ~0.25” of base girth with stretching-only routine.

I have seen examples of erect base girth gains like this with extenders and manual stretching. However, I can’t say for sure how common these types of gains are, except they are much less common than base girth gains from hanging.

I didn’t realize stretcher users were gaining due to the stresses of the noose (that particular thing is new to me).

I mean, I didn’t realize stretcher users were sometimes gaining girth behind the noose.

Originally Posted by blink2000
I mean, I didn’t realize stretcher users were sometimes gaining girth behind the noose.

Is it possible that even prolonged squeezing when using base of glans as a fulcrum to manually stretch the penis also leads to girth gains?


Starting Size: April, 28, 2010: NBPEL-7" Girth-6" (base, MSG, glans)

Currently: BPEL-8" NBPEL-7.25" Girth-6.25" (base)/6.125" (MSG)/6.125" (glans)

Originally Posted by blink2000

I mean, I didn’t realize stretcher users were sometimes gaining girth behind the noose.

I gained a bit of girth there using an extender. Some studies also reported small increases in girth with noose-style extenders. Adding to this that the base girth gains are common expecially among clamp-style hangers users, got me to think that girth gains could be, at least partially, due to ischemic hypertrophy of smooth muscle.

What is ischemic hypertrophy?

Ischemia or hypoxia is reduced blood flow, resulting in lack of oxygenation of a given tissue or organ. Total ischemia is dangerous and leads to necrosis and fibrosis. Partial ischemia, or mild hypoxia, on the other hand, seems to trigger hypertrophy of striatus muscles (f.e. your biceps); it could be that it triggers (benign) hypertrophy in smooth muscles also. This is also the hypothesis of these MDs:

“A case of sickle cell disease with 63 documented
episodes of priapism that were managed
medically is presented. The case is very
unusual because of the fact that despite so
many episodes of priapism, he did not lose
sexual potency. On the contrary, over a period
of time, his penis hypertrophied. To the best of
our knowledge, this is the first such case with
so many episodes of priapism reported in the
English literature. We present a hypothesis for
such unusual occurrence. (J Nati Med Assoc.
1993;85:473-474.)


treatment.
Initial physical examination revealed normal-looking
external genitalia. However, subsequently the penis
gradually showed excessive growth and at the last visit
the penis measured 18 cm in length (from the pubic symphysis
to the tip of the penis) and 161/2 cm in circumference
in flaccid state. The hypertrophy was confined to
the corpora cavernosa. The glans penis and the scrotum
were of normal size. During an episode of priapism, the
penis further increased in size and got very tender.
….
On the one end of the spectrum is total venous occlusion
resulting in priapism, cavernous ischemia and subsequent
fibrosis, and impotence, and on the other end is
the minimal venous occlusion with no priapism and no
ischemic changes. In the middle of the spectrum, there
appears to be various degrees of partial venous
obstruction, resulting in priapism associated with
adequate oxygenation of the cavernous tissue because
of continued circulation in the presence of incomplete
venous occlusion. Our patient represents such an
example. Furthermore, repeated episodes of mild
hypoxia resulting from the transient partial venous
obstruction in our patient might have been a stimulus
for the hypertrophy of corpora cavernosa,
because
hypoxia is known to result in hypertrophy of tissues.”

http://www.ncbi.nlm.nih.gov/pmc/art…a00281-0075.pdf

Many episodes of partial blood flow occlusion: sounds like clamping, doesn’t it? ;)

Just to add to marinera’s post. Although not sure about hypertrophy of dick tissue and while very speculative, some possible mechanisms include the following:

Vascular occlusion leads to intracellular osmole accumulation leads to cell swelling leads to cell membrane/cytoskeleton disruption leads to integrin mechanochemical transduction which finally leads to anabolism-tissue growth. Thus, vascular occlusion and the resulting cell swelling may be seen as a threat to the structural integrity of cell resulting in protein accretion (as a larger muscle cell with more protein would be able to withstand greater internal tension).

Theory-Stimuli for skeletal muscle growth-does it apply to penis


Starting Size: April, 28, 2010: NBPEL-7" Girth-6" (base, MSG, glans)

Currently: BPEL-8" NBPEL-7.25" Girth-6.25" (base)/6.125" (MSG)/6.125" (glans)


Last edited by bohm : 09-27-2010 at .
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